p21Waf1/Cip1 expression and the p53/MDM2 feedback loop in gastric carcinogenesis
- 1 December 1999
- journal article
- research article
- Published by Wiley in The Journal of Pathology
- Vol. 189 (4) , 481-486
- https://doi.org/10.1002/(sici)1096-9896(199912)189:4<481::aid-path482>3.0.co;2-u
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Why is p53 protein stabilized in neoplasia? Some answers but many more questions!The Journal of Pathology, 1998
- Multiple pathways control cell growth and transformation: overlapping and independent activities of p53 and p21Cip1/WAF1/Sdi1The Journal of Pathology, 1997
- Regulation of p53 stability by Mdm2Nature, 1997
- Mdm2 promotes the rapid degradation of p53Nature, 1997
- Cdk-interacting protein 1 directly binds with proliferating cell nuclear antigen and inhibits DNA replication catalyzed by the DNA polymerase delta holoenzyme.Proceedings of the National Academy of Sciences, 1994
- The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinasesCell, 1993
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- The p53-mdm-2 autoregulatory feedback loop.Genes & Development, 1993
- p53 function and dysfunctionCell, 1992
- The mdm-2 oncogene product forms a complex with the p53 protein and inhibits p53-mediated transactivationCell, 1992