Inactivation of the Poly(ADP-ribose) Polymerase Gene Affects Oxygen Radical and Nitric Oxide Toxicity in Islet Cells

This publication has 34 references indexed in Scilit:

• Nitric Oxide Toxicity in Islet Cells Involves Poly(ADP-Ribose) Polymerase Activation and Concomitant NAD+ Depletion
  Biochemical and Biophysical Research Communications, 1994
• Analysis of Oxygen Radical Toxicity in Pancreatic Islets at the Single Cell Level
  Biological Chemistry Hoppe-Seyler, 1994
• Pancreatic islet cells are highly susceptible towards the cytotoxic effects of chemically generated nitric oxide
  Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1993
• Transcription and translation of inducible nitric oxide synthase in the pancreas of prediabetic BB rats
  FEBS Letters, 1993
• Toxicity of chemically generated nitric oxide towards pancreatic islet cells can be prevented by nicotinamide
  Life Sciences, 1992
• Interleukin-1β induces nitric oxide production and inhibits the activity of aconitase without decreasing glucose oxidation rates in isolated mouse pancreatic islets
  Biochemical and Biophysical Research Communications, 1992
• Activated macrophages kill pancreatic syngeneic islet cells via arginine-dependent nitric oxide generation
  Biochemical and Biophysical Research Communications, 1991
• Nicotinamide and 3-aminobenzamide interfere with receptor-mediated transmembrane signaling in murine cytotoxic T cells: Independence of Golgi reorientation from calcium mobilization and inositol phosphate generation
  Cellular Immunology, 1991
• Lymphocyte dysfunction after DNA damage by toxic oxygen species. A model of immunodeficiency.
  The Journal of Experimental Medicine, 1986
• Significance of streptozotocin induced nicotinamide‐adenine‐dinucleotide (NAD) degradation in mouse pancreatic islets
  FEBS Letters, 1973