Influence of Toll-Like Receptor 4, CD14, Tumor Necrosis Factor, and Interleukine-10 Gene Polymorphisms on Clinical Outcome in Japanese Critically Ill Patients
- 31 December 2005
- journal article
- Published by Elsevier in Journal of Surgical Research
- Vol. 129 (2) , 322-328
- https://doi.org/10.1016/j.jss.2005.05.020
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- The immunopathogenesis of sepsisNature, 2002
- GENETIC CONTRIBUTION TO THE SEPTIC RESPONSE IN A MOUSE MODELShock, 2002
- TLR4 mutations are associated with endotoxin hyporesponsiveness in humansNature Genetics, 2000
- C(−260)→T Polymorphism in the Promoter of the CD14 Monocyte Receptor Gene as a Risk Factor for Myocardial InfarctionCirculation, 1999
- A Polymorphism* in the 5 ′ Flanking Region of the CD14 Gene Is Associated with Circulating Soluble CD14 Levels and with Total Serum Immunoglobulin EAmerican Journal of Respiratory Cell and Molecular Biology, 1999
- Endotoxin-tolerant Mice Have Mutations in Toll-like Receptor 4 (Tlr4)The Journal of Experimental Medicine, 1999
- Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 GeneScience, 1998
- Anti-inflammatory cytokine profile and mortality in febrile patientsThe Lancet, 1998
- Antiinflammatory Cytokine Responses during Clinical Sepsis and Experimental Endotoxemia: Sequential Measurements of Plasma Soluble Interleukin (IL)-1 Receptor Type II, IL-10, and IL-13The Journal of Infectious Diseases, 1997
- Plasma tumor necrosis factor and mortality in critically ill septic patientsCritical Care Medicine, 1989