Prion protein fragment 106-126 induces apoptotic cell death and impairment of L-type voltage-sensitive calcium channel activity in the GH3 cell line
- 1 November 1998
- journal article
- research article
- Published by Wiley in Journal of Neuroscience Research
- Vol. 54 (3) , 341-352
- https://doi.org/10.1002/(sici)1097-4547(19981101)54:3<341::aid-jnr5>3.0.co;2-g
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- CREB Phosphorylation and Dephosphorylation: A Ca2+- and Stimulus Duration–Dependent Switch for Hippocampal Gene ExpressionCell, 1996
- Intracellular Calcium Rise through L-Type Calcium Channels, as Molecular Mechanism for Prion Protein Fragment 106-126-Induced Astroglial ProliferationBiochemical and Biophysical Research Communications, 1996
- A neurotoxic prion protein fragment enhances proliferation of microglia but not astrocytes in cultureGlia, 1996
- Role of microglia and host prion protein in neurotoxicity of a prion protein fragmentNature, 1996
- Truncated Forms of the Human Prion Protein in Normal Brain and in Prion DiseasesJournal of Biological Chemistry, 1995
- Neurotoxicity of a prion protein fragmentNature, 1993
- Scrapie-associated prion protein accumulates in astrocytes during scrapie infection.Proceedings of the National Academy of Sciences, 1991
- Calcium current in cortical astrocytes: induction by cAMP and neurotransmitters and permissive effect of serum factorsJournal of Neuroscience, 1989
- Precise targeting of the pathology of the sialoglycoprotein, PrP, and vacuolar degeneration in mouse scrapieNeuroscience Letters, 1989
- Changes in the localization of brain prion proteins during scrapie infectionNeurology, 1987