ApoAI Deficiency Results in Marked Reductions in Plasma Cholesterol But No Alterations in Amyloid-β Pathology in a Mouse Model of Alzheimer's Disease-Like Cerebral Amyloidosis

This publication has 82 references indexed in Scilit:

• Brain region-dependent increases in ?-amyloid and apolipoprotein E levels in hypercholesterolemic rabbits
  Journal Of Neural Transmission-Parkinsons Disease and Dementia Section, 2003
• Differences in the A?40/A?42 ratio associated with cerebrospinal fluid lipoproteins as a function of apolipoprotein E genotype
  Annals of Neurology, 2000
• Decreased high-density lipoprotein cholesterol and serum apolipoprotein AI concentrations are highly correlated with the severity of Alzheimer’s disease☆
  Neurobiology of Aging, 2000
• Elevated Low-Density Lipoprotein in Alzheimer's Disease Correlates with Brain Aβ 1–42 Levels
  Biochemical and Biophysical Research Communications, 1998
• Biochemical Characterization of Alzheimer's Soluble Amyloid Beta Protein in Human Cerebrospinal Fluid: Association with High Density Lipoproteins
  Biochemical and Biophysical Research Communications, 1996
• The Soluble Form of Alzheimer′s Amyloid β Protein Is Complexed to High Density Lipoprotein 3 and Very High Density Lipoprotein in Normal Human Plasma
  Biochemical and Biophysical Research Communications, 1994
• Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease.
  Proceedings of the National Academy of Sciences, 1993
• Pathophysiology of reverse cholesterol transport. Insights from inherited disorders of lipoprotein metabolism.
  Arteriosclerosis: An Official Journal of the American Heart Association, Inc., 1989
• A DNA insertion in the apolipoprotein A-I gene of patients with premature atherosclerosis
  Nature, 1983
• Familial Deficiency of Apolipoproteins A-I and C-III and Precocious Coronary-Artery Disease
  New England Journal of Medicine, 1982