Nε(γ-glutamyl)lysine in cerebrospinal fluid marks Alzheimer type and vascular dementia
- 30 June 2001
- journal article
- research article
- Published by Elsevier in Neurobiology of Aging
- Vol. 22 (3) , 403-406
- https://doi.org/10.1016/s0197-4580(01)00224-x
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Critically attained threshold of cerebral hypoperfusion: the CATCH hypothesis of Alzheimer’s pathogenesisNeurobiology of Aging, 2000
- The role of cerebral ischemia in Alzheimer’s diseaseNeurobiology of Aging, 2000
- Differential Expression of Multiple Transglutaminases in Human BrainPublished by Elsevier ,1999
- `Tissue' transglutaminase in cell death: a downstream or a multifunctional upstream effector?FEBS Letters, 1998
- Induction of "tissue" transglutaminase in HIV pathogenesis: evidence for high rate of apoptosis of CD4+ T lymphocytes and accessory cells in lymphoid tissues.Proceedings of the National Academy of Sciences, 1996
- Alteration of transglutaminase activity in rat and human spinal cord after neuronal degenerationNeurochemical Research, 1995
- Reliability of NINDS‐AIREN clinical criteria for the diagnosis of vascular dementiaNeurology, 1994
- Presence and Possible Origin of ɛ(γ-Glutamyl)Lysine Isodipeptide in Human PlasmaThrombosis and Haemostasis, 1992
- Degradation of cells dying by apoptosis leads to accumulation of ε(γ‐glutamyl)lysine isodipeptide in culture fluid and bloodFEBS Letters, 1991
- ?-Glutamylamine cyclotransferaseMolecular and Cellular Biochemistry, 1981