Compensatory renal hypertrophy is mediated by a cell cycle-dependent mechanism
- 1 November 2002
- journal article
- Published by Elsevier in Kidney International
- Vol. 62 (5) , 1650-1658
- https://doi.org/10.1046/j.1523-1755.2002.00620.x
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- High Glucose-Induced Hypertrophy of Mesangial Cells Requires p27Kip1, an Inhibitor of Cyclin-Dependent KinasesThe American Journal of Pathology, 2001
- G1 kinases and transforming growth factor-β; signaling are associated with a growth pattern switch in diabetes-induced renal growthKidney International, 2000
- What Makes Cells Grow Larger and How Do They Do It? Renal Hypertrophy RevisitedNephron Experimental Nephrology, 1999
- Glomerular expression of p27Kip1 in diabetic db/db mouse: Role of hyperglycemiaKidney International, 1998
- Inhibition of Cyclin‐Dependent Kinases by Purine AnaloguesEuropean Journal of Biochemistry, 1997
- Angiotensin II-stimulated hypertrophy of LLC-PK1 cells depends on the induction of the cyclin-dependent kinase inhibitor p27Kip1Kidney International, 1996
- Influence of ammonia and pH on protein and amino acid metabolism in LLC-PK1 cellsKidney International, 1992
- Retinoblastoma protein switches the E2F site from positive to negative elementNature, 1992
- Increased Na/H antiporter and Na/3HCO3 symporter activities in chronic hyperfiltration. A model of cell hypertrophy.The Journal of general physiology, 1991
- 23Na NMR spectroscopy of proximal tubule suspensionsKidney International, 1986