Incidence and persistence of 8-oxo-7,8-dihydroguanine within a hairpin intermediate exacerbates a toxic oxidation cycle associated with trinucleotide repeat expansion
- 2 July 2011
- journal article
- research article
- Published by Elsevier in DNA Repair
- Vol. 10 (8) , 887-896
- https://doi.org/10.1016/j.dnarep.2011.06.003
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Substrate Channeling in Mammalian Base Excision Repair Pathways: Passing the BatonJournal of Biological Chemistry, 2010
- Mechanisms of trinucleotide repeat instability during human developmentNature Reviews Genetics, 2010
- Replication-dependent instability at (CTG)•(CAG) repeat hairpins in human cellsNature Chemical Biology, 2010
- Trinucleotide repeats in human genome and exomeNucleic Acids Research, 2010
- Stoichiometry of Base Excision Repair Proteins Correlates with Increased Somatic CAG Instability in Striatum over Cerebellum in Huntington's Disease Transgenic MicePLoS Genetics, 2009
- Coordination between Polymerase β and FEN1 Can Modulate CAG Repeat ExpansionJournal of Biological Chemistry, 2009
- Base-excision repair of oxidative DNA damageNature, 2007
- OGG1 initiates age-dependent CAG trinucleotide expansion in somatic cellsNature, 2007
- Huntington's disease: new hope for therapeuticsTrends in Neurosciences, 2001
- Optical properties of sixteen dinucleoside phosphatesJournal of Molecular Biology, 1966