Differential regulation of the CXCR2 chemokine network in rat brain trauma: Implications for neuroimmune interactions and neuronal survival
- 1 May 2006
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 22 (2) , 312-322
- https://doi.org/10.1016/j.nbd.2005.11.015
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- N-Methyl-D-aspartate Attenuates CXCR2-Mediated Neuroprotection through Enhancing the Receptor Phosphorylation and Blocking the Receptor RecyclingMolecular Pharmacology, 2005
- Spatial and Temporal Characteristics of Neurodegeneration after Controlled Cortical Impact in Mice: More than a Focal Brain InjuryJournal of Neurotrauma, 2005
- Circulating monocytic cells infiltrate layers of anterograde axonal degeneration where they transform into microgliaThe FASEB Journal, 2005
- Low nuclear levels of nuclear factor-?B are essential for KC self-induction in astrocytes: Requirements for shuttling and phosphorylationGlia, 2004
- On the Mechanism and Significance of Ligand-induced Internalization of Human Neutrophil Chemokine Receptors CXCR1 and CXCR2Journal of Biological Chemistry, 2004
- Bone marrow stem cells have the ability to populate the entire central nervous system into fully differentiated parenchymal microgliaThe FASEB Journal, 2004
- Widely expressed transcripts for chemokine receptor CXCR1 in identified glutamatergic, γ‐aminobutyric acidergic, and cholinergic neurons and astrocytes of the rat brain: A single‐cell reverse transcription‐multiplex polymerase chain reaction studyJournal of Neuroscience Research, 2003
- Modification of microglia function protects from lesion‐induced neuronal alterations and promotes sprouting in the hippocampusThe FASEB Journal, 2003
- Novel therapies in development for the treatment of traumatic brain injuryExpert Opinion on Investigational Drugs, 2002
- Role of Neutrophils in Radical Production during Ischemia and Reperfusion of the Rat Brain: Effect of Neutrophil Depletion on Extracellular Ascorbyl Radical FormationJournal of Cerebral Blood Flow & Metabolism, 1995