Blood monocyte oxidative burst activity in acute P. falciparum malaria
- 1 January 1989
- Vol. 97 (1-6) , 469-471
- https://doi.org/10.1111/j.1699-0463.1989.tb00817.x
Abstract
The release of superoxide anion from blood monocytes was studied in eight patients with acute primary attack P. falciparum malaria. Before treatment a significant enhancement of the oxidative burst prevailed, which contrasts with previous findings of a depressed monocyte chemotactic responsiveness. During treatment and after clinical recovery the activity of superoxide anion release normalized in all patients.Keywords
This publication has 20 references indexed in Scilit:
- Generation of Reactive Oxygen Radicals by Human Phagocytic Cells Activated by Plasmodium falciparumScandinavian Journal of Immunology, 1987
- Suppression of blood monocyte and neutrophil chemotaxis in acute human malariaParasite Immunology, 1986
- Induction of crisis forms in the human malaria parasite Plasmodium falciparum by gamma-interferon-activated, monocyte-derived macrophages.The Journal of Immunology, 1984
- Oxidative killing of the intraerythrocytic malaria parasite Plasmodium yoelii by activated macrophages.The Journal of Immunology, 1984
- N-formylmethionyl-leucyl-[3H]phenylalanine binding, superoxide release, and chemotactic responses of human blood monocytes that repopulate the circulation during leukapheresisBlood, 1983
- Differences in superoxide production by nonmigrating and migrating human monocyte subpopulationsBiochemical and Biophysical Research Communications, 1982
- Biological Defense Mechanisms. THE PRODUCTION BY LEUKOCYTES OF SUPEROXIDE, A POTENTIAL BACTERICIDAL AGENTJournal of Clinical Investigation, 1973
- Cytochemical Identification of Monocytes and GranulocytesAmerican Journal of Clinical Pathology, 1971