Tpl2/Cot Signals Activate ERK, JNK, and NF-κB in a Cell-type and Stimulus-specific Manner
Open Access
- 1 June 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (25) , 23748-23757
- https://doi.org/10.1074/jbc.m412837200
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- IκB Kinase Is an Essential Component of the Tpl2 Signaling PathwayMolecular and Cellular Biology, 2004
- Differential regulation of interleukin 1 receptor and Toll-like receptor signaling by MEKK3Nature Immunology, 2003
- Anti-TNF-α therapies: the next generationNature Reviews Drug Discovery, 2003
- Tumor necrosis factor signalingCell Death & Differentiation, 2003
- Phosphorylation of serine 276 is essential for p65 NF- B subunit-dependent cellular responsesBiochemical and Biophysical Research Communications, 2003
- TNF-R1 Signaling: A Beautiful PathwayScience, 2002
- The Oncogenic Protein Kinase Tpl-2/Cot Contributes to Epstein-Barr Virus-Encoded Latent Infection Membrane Protein 1-Induced NF-κB Signaling Downstream of TRAF2Journal of Virology, 2002
- Tpl-2 induces IL-2 expression in T-cell lines by triggering multiple signaling pathways that activate NFAT and NF-κBOncogene, 1998
- Phosphorylation of NF-κB p65 by PKA Stimulates Transcriptional Activity by Promoting a Novel Bivalent Interaction with the Coactivator CBP/p300Molecular Cell, 1998
- The Transcriptional Activity of NF-κB Is Regulated by the IκB-Associated PKAc Subunit through a Cyclic AMP–Independent MechanismCell, 1997