Pergolide Presynaptically Inhibits Calcium‐Stimulated Release of γ‐Aminobutyric Acid

Abstract
The release of γ‐aminobutyric acid (GABA) in rat dorsolateral striatum was studied using in vivo microdialysis. Dialysis was conducted 2 days after probe implantation in awake, freely moving rats using a modified Ringer solution. Calcium induced a reversible increase in GABA release that was abolished by tetrodotoxin but was only slightly attenuated by a maximally effective dose of pergolide, a D2 receptor agonist. It was thus concluded that pergolide inhibits calcium‐stimulated release of GABA presynaptically by a mechanism distinct from that of tetrodotoxin.