Bcl-2 family-mediated apoptotic effects of 3,3′-diindolylmethane (DIM) in human breast cancer cells
- 1 March 2002
- journal article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 63 (6) , 1085-1097
- https://doi.org/10.1016/s0006-2952(02)00856-0
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Translocation of Bax to mitochondria induces apoptotic cell death in Indole-3-carbinol (I3C) treated breast cancer cellsOncogene, 2000
- The Bcl-2 Protein FamilyExperimental Cell Research, 2000
- Aryl hydrocarbon receptor-mediated antiestrogenic and antitumorigenic activity of diindolylmethaneCarcinogenesis: Integrative Cancer Research, 1998
- Overexpression of the death-promoting gene bax-alpha which is downregulated in breast cancer restores sensitivity to different apoptotic stimuli and reduces tumor growth in SCID mice.Journal of Clinical Investigation, 1996
- Expression of the bcl‐2 gene family in normal and malignant breast tissue: Low bax‐α expression in tumor cells correlates with resistance towards apoptosisInternational Journal of Cancer, 1995
- Calpain activation in apoptosisJournal of Cellular Physiology, 1994
- Bcl-2 and the regulation of programmed cell deathThe Journal of cell biology, 1994
- Effects of dietary indole-3-carbinol on estradiol metabolism and spontaneous mammary tumors in miceCarcinogenesis: Integrative Cancer Research, 1991
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990
- Structure—activity relationships of dietary indoles: A proposed mechanism of action as modifiers of xenobiotic metabolismJournal of Toxicology and Environmental Health, 1987