Perinatal Hypoxia and Bioelectric Brain Maturation of the Newborn Infant1

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Abstract
Bioelectric brain maturation of 20 infants who had suffered acute perinatal hypoxia (patients) was compared with that of 20 healthy newborns (controls). None of the patients had suffered any other pre- or perinatal complications that could have influenced the bioelectric brain maturation. All infants (postmenstrual age: 40-42 wk) were subjected to a polygraphic recording. The patients were examined after the acute phase of their disease; all were in good clinical condition at the time of recording. Statistical evaluation revealed significantly retarded bioelectric brain maturation in the patient group. A more immature EEG pattern corresponded to greater extent of O2 deprivation. Determination of bioelectric brain maturation can be used to obtain information about hypoxia and the extent of O2 deprivation.

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