Pertussis toxin inhibits the rise in the intracellular concentration of free calcium that is induced by chemotactic factors in rabbit neutrophils: Possible role of the “G proteins” in calcium mobilization
- 30 October 1984
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 124 (2) , 644-650
- https://doi.org/10.1016/0006-291x(84)91603-6
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Effect of pertussis toxin on α2 -adrenoceptors: decreased formation of the high-affinity state for agonistsFEBS Letters, 1984
- Inositol trisphosphate and diacylglycerol as second messengersBiochemical Journal, 1984
- G proteins and dual control of adenylate cyclaseCell, 1984
- Involvement of guanine nucleotide-binding protein in the gating of Ca2+ by receptorsNature, 1983
- Guanine nucleotides modulate the binding affinity of the oligopeptide chemoattractant receptor on human polymorphonuclear leukocytes.Journal of Clinical Investigation, 1983
- The cytoplasmic concentration of free calcium in platelets is controlled by stimulators of cyclic AMP production (PGD2, PGE1, forskolin)Biochemical and Biophysical Research Communications, 1983
- Islet‐activating protein prevents nicotinic acid‐induced GTPase stimulation and GTP but not GTPγS‐induced adenylate cyclase inhibition in rat adipocytesFEBS Letters, 1983
- Direct demonstration of increased intracellular concentration of free calcium in rabbit and human neutrophils following stimulation by chemotactic factorBiochemical and Biophysical Research Communications, 1983
- Tumour promoter uncouples β-adrenergic receptor from adenyl cyclase in mouse epidermisNature, 1980
- Effect of chemotactic factors on calcium levels of rabbit neutrophilsAmerican Journal of Physiology-Cell Physiology, 1979