Signal Transduction Pathways in Hyperoxia-Induced Lung Cell Death
Open Access
- 1 September 2000
- journal article
- review article
- Published by Elsevier in Molecular Genetics and Metabolism
- Vol. 71 (1-2) , 359-370
- https://doi.org/10.1006/mgme.2000.3046
Abstract
No abstract availableKeywords
This publication has 89 references indexed in Scilit:
- Cell density and antioxidant vitamins determine the effects of hyperoxia on proliferation and death of MDCK epithelial cellsNutrition and Cancer, 1997
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996
- Persistent Activation of c-Jun N-terminal Kinase 1 (JNK1) in γ Radiation-induced ApoptosisJournal of Biological Chemistry, 1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995
- Requirement for MAP Kinase (ERK2) Activity in Interferon α- and Interferon β-Stimulated Gene Expression Through STAT ProteinsScience, 1995
- Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death.The Journal of Experimental Medicine, 1995
- Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and ThreonineJournal of Biological Chemistry, 1995
- Fas and tumor necrosis factor receptor-mediated cell death: similarities and distinctions.The Journal of Experimental Medicine, 1994
- Antioxidant status and lipid peroxidation in patients infected with HIVChemico-Biological Interactions, 1994