Adenosine A1 receptors inhibit both adenylate cyclase activity and TRH-activated Ca2+ channels by a pertussis toxin-sensitive mechanism in GH3 cells

This publication has 32 references indexed in Scilit:

• Oscillations of cytosolic Ca2+ in pituitary cells due to action potentials
  Nature, 1987
• Vasoactive intestinal peptide and peptide with N-terminal histidine and C-terminal isoleucine increase prolactin secretion in cultured rat pituitary cells (GE4C1) via a cAMP-dependent mechanism which involves transient elevation of intracellular Ca2+
  Molecular and Cellular Endocrinology, 1987
• Micro-injection of inositol 1,3,4,5-tetrakisphosphate activates sea urchin eggs by a mechanism dependent on external Ca2+
  Biochemical Journal, 1986
• Pertussis toxin selectively abolishes hormone induced lowering of cytosolic calcium in GH₃ cells
  FEBS Letters, 1985
• Thyrotropin-releasing hormone increases cytosolic free Ca2+ in clonal pituitary cells (GH3 cells): direct evidence for the mobilization of cellular calcium.
  The Journal of cell biology, 1984
• The Somatostatin Receptor Is Directly Coupled to Adenylate Cyclase in GH4Ci Pituitary Cell Membranes*
  Endocrinology, 1984
• Calcium channel modulation by neurotransmitters, enzymes and drugs
  Nature, 1983
• Vasoactive intestinal peptide (VIP) stimulates prolactin (PRL) release and cAMP production in a rat pituitary cell line (GH3/B6). Additive effects of VIP and TRH on PRL release
  FEBS Letters, 1979
• Membrane Effects of Thyrotropin-Releasing Hormone and Estrogen Shown by Intracellular Recording from Pituitary Cells
  Science, 1979
• [46] Clonal strains of hormone-producing pituitary cells
  Published by Elsevier ,1979