The stabilization of repetitive tracts of DNA by variant repeats requires a functional DNA mismatch repair system
- 1 November 1995
- Vol. 83 (4) , 539-545
- https://doi.org/10.1016/0092-8674(95)90093-4
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancerCell, 1995
- Gametic and somatic tissue–specific heterogeneity of the expanded SCA1 CAG repeat in spinocerebellar ataxia type 1Nature Genetics, 1995
- Trinucleotide repeats that expand in human disease form hairpin structures in vitroCell, 1995
- Length of uninterrupted CGG repeats determines instability in the FMR1 geneNature Genetics, 1994
- Instability of simple sequence repeats in a mammalian cell lineHuman Molecular Genetics, 1994
- Evidence for a mechanism predisposing to intergenerational CAG repeat instability in spinocerebellar ataxia type INature Genetics, 1993
- Transposon Tn5 excision in yeast: influence of DNA polymerases alpha, delta, and epsilon and repair genes.Proceedings of the National Academy of Sciences, 1992
- Measurements of excision repair tracts formed during meiotic recombination in Saccharomyces cerevisiae.Molecular and Cellular Biology, 1992
- [12] High-efficiency transformation of yeast by electroporationPublished by Elsevier ,1991
- A positive selection for mutants lacking orotidine-5′-phosphate decarboxylase activity in yeast: 5-fluoro-orotic acid resistanceMolecular Genetics and Genomics, 1984