Early reduction of NeuN antigenicity induced by soman poisoning in mice can be used to predict delayed neuronal degeneration in the hippocampus

This publication has 19 references indexed in Scilit:

• Neuronal regeneration partially compensates the delayed neuronal cell death observed in the hippocampal CA1 field of soman-poisoned mice
  NeuroToxicology, 2005
• Modulation of neurodegeneration by molecular chaperones
  Nature Reviews Neuroscience, 2005
• Chaperones, protein aggregation, and brain protection from hypoxic/ischemic injury
  Journal of Experimental Biology, 2004
• Loss of NeuN immunoreactivity after cerebral ischemia does not indicate neuronal cell loss: a cautionary note
  Brain Research, 2004
• Effects of Global Ischemia Duration on Neuronal, Astroglial, Oligodendroglial, and Microglial Reactions in the Vulnerable Hippocampal CA1 Subregion in Rats
  Journal of Neurotrauma, 2002
• Regional Vulnerability after Traumatic Brain Injury: Gender Differences in Mice That Overexpress Human Copper, Zinc Superoxide Dismutase
  Experimental Neurology, 2001
• Effects of Thienylphencyclidine (TCP) on Seizure Activity and Brain Damage Produced by Soman in Guinea-Pigs: ECoG Correlates of Neurotoxicity
  NeuroToxicology, 2001
• Attenuation of Delayed Neuronal Death after Mild Focal Ischemia in Mice by Inhibition of the Caspase Family
  Journal of Cerebral Blood Flow & Metabolism, 1998
• The Role of Glutamate Neurotoxicity in Hypoxic-Ischemic Neuronal Death
  Annual Review of Neuroscience, 1990
• Histological and histochemical changes in the central nervous system of the rat poisoned by an irreversible anticholinesterase organophosphorus compound
  Acta Neuropathologica, 1983