In utero cocaine exposure decreases dopamine D1 receptor modulation of hippocampal long-term potentiation in the rabbit
- 13 September 1996
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 215 (3) , 157-160
- https://doi.org/10.1016/0304-3940(96)12960-8
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Prenatal cocaine exposure leads to enhanced long-term potentiation in region CA1 of hippocampusDevelopmental Brain Research, 1996
- Effects of prenatal cocaine exposure upon postnatal development of neostriatal dopaminergic functionSynapse, 1994
- Serotonin controls the magnitude of LTP induced by theta bursts via an action on NMDA-receptor-mediated responsesBrain Research, 1994
- Prenatal cocaine produces deficits in serotonin mediated neuroendocrine responses in adult rat progeny: Evidence for long‐term functional alterations in brain serotonin pathwaysSynapse, 1993
- Body weight gain and hippocampal volumes of rats exposed neonatally to psychostimulantsBrain Research, 1993
- Effects of cAMP Simulate a Late Stage of LTP in Hippocampal CA1 NeuronsScience, 1993
- Different laminar distributions of dopamine D1 and D2 receptors in the rat hippocampal regionNeuroscience Letters, 1991
- Structural and functional effects of prenatal cocaine exposure in adult rat brainDevelopmental Brain Research, 1990
- Weanling rats exposed prenatally to cocaine exhibit an increase in striatal D2 dopamine binding associated with an increase in ligand affinityPharmacology Biochemistry and Behavior, 1990
- Distribution of dopamine D1 and D2 receptors in rabbit cortical areas, hippocampus, and neostriatum in relation to dopamine contentsSynapse, 1988