Prolonged blockade of nitric oxide synthesis in gravid rats produces sustained hypertension, proteinuria, thrombocytopenia, and intrauterine growth retardation
- 1 May 1994
- journal article
- research article
- Published by Elsevier in American Journal of Obstetrics and Gynecology
- Vol. 170 (5) , 1458-1466
- https://doi.org/10.1016/s0002-9378(94)70179-2
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Nω-Nitro-L-arginine, an inhibitor of nitric oxide synthesis, increases blood pressure in rats and reverses the pregnancy-induced refractoriness to vasopressor agentsAmerican Journal of Obstetrics and Gynecology, 1992
- Adverse Interactions Between Pregnancy and a New Model of Systemic Hypertension Produced by Chronic Blockade of Endothelial Derived Relaxing Factor (EDRF) in the RatClinical and Experimental Hypertension. Part B: Hypertension in Pregnancy, 1992
- Nitric oxide. A novel signal transduction mechanism for transcellular communication.Hypertension, 1990
- Pressor responsiveness to endothelin is not attenuated in gravid ratsLife Sciences, 1990
- Vascular endothelial cells synthesize nitric oxide from L-arginineNature, 1988
- Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factorNature, 1987
- Modulation of Vascular Smooth Muscle Contraction by the EndotheliumAnnual Review of Physiology, 1986
- Mesangial immune injury, hypertension, and progressive glomerular damage in Dahl ratsKidney International, 1984
- The plasma, cell and blood volumes of albino rabbits as estimated by the dye (T 1824) and 32P marked cell methodsThe Journal of Physiology, 1952
- Morphology and Enumeration of Human Blood PlateletsJournal of Applied Physiology, 1950