CCR2-/- knockout mice revascularize normally in response to severe hindlimb ischemia
- 24 August 2004
- journal article
- Published by Elsevier in Journal of Vascular Surgery
- Vol. 40 (4) , 786-795
- https://doi.org/10.1016/j.jvs.2004.07.012
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Arteriogenesis Proceeds via ICAM-1/Mac-1- Mediated MechanismsCirculation Research, 2004
- Transplantation of Monocytes: A Novel Strategy forIn VivoAugmentation of Collateral Vessel GrowthHuman Gene Therapy, 2004
- Factors Regulating ArteriogenesisArteriosclerosis, Thrombosis, and Vascular Biology, 2003
- VEGFR-1–Selective VEGF Homologue PlGF Is ArteriogenicCirculation Research, 2003
- Local Monocyte Chemoattractant Protein-1 Therapy Increases Collateral Artery Formation in Apolipoprotein E–Deficient Mice but Induces Systemic Monocytic CD11b Expression, Neointimal Formation, and Plaque ProgressionCirculation Research, 2003
- Blood monocyte concentration is critical for enhancement of collateral artery growthAmerican Journal of Physiology-Heart and Circulatory Physiology, 2002
- Nerve growth factor induces angiogenic activity in a mouse model of hindlimb ischemiaNeuroscience Letters, 2002
- GM-CSF: a strong arteriogenic factor acting by amplification of monocyte functionAtherosclerosis, 2001
- Local Delivery of Human Tissue Kallikrein Gene Accelerates Spontaneous Angiogenesis in Mouse Model of Hindlimb IschemiaCirculation, 2001
- Monocyte Chemotactic Protein-1 Increases Collateral and Peripheral Conductance After Femoral Artery OcclusionCirculation Research, 1997