Neuronal Intranuclear Inclusions in Polyglutamine Diseases: Nuclear Weapons or Nuclear Fallout?

This publication has 19 references indexed in Scilit:

• Huntingtin Acts in the Nucleus to Induce Apoptosis but Death Does Not Correlate with the Formation of Intranuclear Inclusions
  Cell, 1998
• SH3GL3 Associates with the Huntingtin Exon 1 Protein and Promotes the Formation of Polygln-Containing Protein Aggregates
  Molecular Cell, 1998
• Ataxin-1 Nuclear Localization and Aggregation
  Cell, 1998
• Suppression of aggregate formation and apoptosis by transglutaminase inhibitors in cells expressing truncated DRPLA protein with an expanded polyglutamine stretch
  Nature Genetics, 1998
• Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat disorders with polyglutamine-repeat expansions?
  The Lancet, 1998
• Intranuclear Neuronal Inclusions: A Common Pathogenic Mechanism for Glutamine-Repeat Neurodegenerative Diseases?
  Published by Elsevier ,1997
• Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in Brain
  Science, 1997
• Peptides containing glutamine repeats as substrates for transglutaminase-catalyzed cross-linking: Relevance to diseases of the nervous system
  Proceedings of the National Academy of Sciences, 1996
• Cleavage of huntingtin by apopain, a proapoptotic cysteine protease, is modulated by the polyglutamine tract
  Nature Genetics, 1996
• Glutamine repeats as polar zippers: their possible role in inherited neurodegenerative diseases.
  Proceedings of the National Academy of Sciences, 1994