Thrombin Induces Nitric-oxide Synthase via Gα12/13-coupled Protein Kinase C-dependent I-κBα Phosphorylation and JNK-mediated I-κBα Degradation

This publication has 53 references indexed in Scilit:

• Activation of the fibrinolytic system and utilization of the coagulation inhibitors in sepsis: comparison with severe sepsis and septic shock
  Intensive Care Medicine, 2001
• Coagulation and inflammation
  Innate Immunity, 2001
• Endotoxaemia in patients with severe sepsis or septic shock.
  Intensive Care Medicine, 2000
• Regulation of blood coagulation
  Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 2000
• Cellular origin and procoagulant properties of microparticles in meningococcal sepsis
  Blood, 2000
• Coagulation system and platelets are fully activated in uncomplicated sepsis
  Critical Care Medicine, 2000
• Known bacterial virulence factors do not explain the variation in urinary cytokine levels in patients with urosepsis
  FEMS Immunology & Medical Microbiology, 1996
• Regulation and Function of Inducible Nitric Oxide Synthase during Sepsis and Acute Inflammation
  Published by Elsevier ,1995
• Attenuation of the induction of nitric oxide synthase by endogenous glucocorticoids accounts for endotoxin tolerance in vivo.
  Proceedings of the National Academy of Sciences, 1994
• Coagulation, fibrinolysis, and kallikrein systems in sepsis
  Critical Care Medicine, 1989