Effect of protein kinase C blockade on phosphorylation of NR1 in dorsal horn and spinothalamic tract cells caused by intradermal capsaicin injection in rats
- 1 September 2004
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 1020 (1-2) , 95-105
- https://doi.org/10.1016/j.brainres.2004.06.017
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Inhibition of protein kinase C reduces ischemia-induced tyrosine phosphorylation of the N-methyl-d-aspartate receptor.Journal of Neurochemistry, 2003
- Calcium–Calmodulin-Dependent Protein Kinase II Contributes to Spinal Cord Central SensitizationJournal of Neuroscience, 2002
- Chronic Hypersensitivity For Inflammatory Nociceptor Sensitization Mediated by the ε Isozyme of Protein Kinase CJournal of Neuroscience, 2000
- N-Methyl-d-aspartate receptors are present in vagal afferents and their dendritic targets in the nucleus tractus solitariusNeuroscience, 1999
- A novel heat-activated current in nociceptive neurons and its sensitization by bradykininProceedings of the National Academy of Sciences, 1996
- Enhancement of the response in spinal dorsal horn neurons by cAMP-dependent protein kinaseNeuroscience Letters, 1993
- Cyclic adenosine 3′5′-monophosphate potentiates excitatory amino acid and synaptic responses of rat spinal dorsal horn neuronsBrain Research, 1992
- Protein kinase C reduces Mg2+ block of NMDA-receptor channels as a mechanism of modulationNature, 1992
- Activity-dependent neuronal plasticity following tissue injury and inflammationTrends in Neurosciences, 1992
- Sustained potentiation of NMDA receptor-mediated glutamate responses through activation of protein kinase C by a μ opioidNeuron, 1991