Therapeutic options to minimize free radical damage and thrombogenicity in ischemic/reperfused myocardium

This publication has 72 references indexed in Scilit:

• Reperfusion injury: laboratory artifact or clinical dilemma?
  Circulation, 1986
• Myocardial ischemia revisited. The osmolar load, membrane damage, and reperfusion
  Journal of Molecular and Cellular Cardiology, 1986
• Free radicals and myocardial injury: pharmacologic implications.
  Circulation, 1986
• The oxygen free radical system: A fundamental mechanism in the production of myocardial necrosis
  Progress in Cardiovascular Diseases, 1986
• Oxygen-mediated myocardial damage during ischameia and reperfusion: Role of the cellular defences against oxygen toxicity
  Journal of Molecular and Cellular Cardiology, 1985
• The oxygen free radical system: Potential mediator of myocardial injury
  Journal of the American College of Cardiology, 1985
• Characterization of cardiac sarcoplasmic reticulum dysfunction during short-term, normothermic, global ischemia.
  Circulation Research, 1984
• In vitro study on contribution of oxidative metabolism of isolated rabbit heart mitochondria to myocardial reperfusion injury.
  Circulation Research, 1984
• Free radical mediation of the effects of acidosis on calcium transport by cardiac sarcoplasmic reticulum in whole heart homogenates
  Cardiovascular Research, 1984
• The role of lipid peroxidation in pathogenesis of ischemic damage and the antioxidant protection of the heart
  Basic Research in Cardiology, 1982