The Mre11 complex and the metabolism of chromosome breaks: the importance of communicating and holding things together
- 1 August 2004
- journal article
- review article
- Published by Elsevier in DNA Repair
- Vol. 3 (8-9) , 845-854
- https://doi.org/10.1016/j.dnarep.2004.03.014
Abstract
No abstract availableKeywords
This publication has 80 references indexed in Scilit:
- DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociationNature, 2003
- Interaction of FANCD2 and NBS1 in the DNA damage responseNature Cell Biology, 2002
- NBS1 Localizes to γ-H2AX Foci through Interaction with the FHA/BRCT DomainCurrent Biology, 2002
- Nbs1 promotes ATM dependent phosphorylation events including those required for G1/S arrestOncogene, 2002
- A Murine Model of Nijmegen Breakage SyndromeCurrent Biology, 2002
- Nijmegen breakage syndrome: Clinical characteristics and mutation analysis in eight unrelated Russian familiesThe Journal of Pediatrics, 2002
- Human Rad50/Mre11 Is a Flexible Complex that Can Tether DNA EndsMolecular Cell, 2001
- Chk2 Activation Dependence on Nbs1 after DNA DamageMolecular and Cellular Biology, 2001
- Distinct Functional Domains of Nibrin Mediate Mre11 Binding, Focus Formation, and Nuclear LocalizationMolecular and Cellular Biology, 2001
- Meiosis-Specific DNA Double-Strand Breaks Are Catalyzed by Spo11, a Member of a Widely Conserved Protein FamilyCell, 1997