A Reciprocal Interdependence between Nck and PI(4,5)P2 Promotes Localized N-WASp-Mediated Actin Polymerization in Living Cells
- 1 November 2009
- journal article
- research article
- Published by Elsevier in Molecular Cell
- Vol. 36 (3) , 525-535
- https://doi.org/10.1016/j.molcel.2009.10.025
Abstract
No abstract availableKeywords
Funding Information
- American Heart Association (0735252N)
- National Institutes of Health
This publication has 30 references indexed in Scilit:
- Clustering of Nck by a 12-residue Tir phosphopeptide is sufficient to trigger localized actin assemblyThe Journal of cell biology, 2004
- Inducible Clustering of Membrane-Targeted SH3 Domains of the Adaptor Protein Nck Triggers Localized Actin PolymerizationCurrent Biology, 2004
- Specificity in Signal TransductionCell, 2004
- The Murine Nck SH2/SH3 Adaptors Are Important for the Development of Mesoderm-Derived Embryonic Structures and for Regulating the Cellular Actin NetworkMolecular and Cellular Biology, 2003
- Phosphatidylinositol 4,5-Biphosphate (PIP2)-induced Vesicle Movement Depends on N-WASP and Involves Nck, WIP, and Grb2Journal of Biological Chemistry, 2002
- Visualizing Cellular Phosphoinositide Pools with GFP-Fused Protein-ModulesScience's STKE, 2002
- The large GTPase dynamin regulates actin comet formation and movement in living cellsProceedings of the National Academy of Sciences, 2002
- Nck/Dock: an adapter between cell surface receptors and the actin cytoskeletonOncogene, 2001
- Enteropathogenic E. coli Tir binds Nck to initiate actin pedestal formation in host cellsNature Cell Biology, 2001
- Nck and Phosphatidylinositol 4,5-Bisphosphate Synergistically Activate Actin Polymerization through the N-WASP-Arp2/3 PathwayJournal of Biological Chemistry, 2001