Intracellular calcium concentration impairment in hepatocytes from thioacetamide-treated rats. Implications for the activity of Ca2+-dependent enzymes

This publication has 35 references indexed in Scilit:

• Relationship between genomic DNA ploidy and parameters of liver damage during necrosis and regeneration induced by thioacetamide
  Hepatology, 1993
• Isoenzymes of Carbohydrate Metabolism in Primary Cultures of Hepatocytes From Thioacetamide–Induced Rat Liver Necrosis: Responses to Growth Factors
  Hepatology, 1992
• Review of the current status of calcium and thiols in cellular injury
  Chemical Research in Toxicology, 1990
• On the role of calcium in the toxicity of alkylating and oxidizing quinone imines in isolated hepatocytes
  Chemical Research in Toxicology, 1989
• Cells without growth factors commit suicide
  Science, 1988
• Effect of acetaminophen hepatotoxicity on hepatic mitochondrial and microsomal calcium contents in mice
  Toxicology Letters, 1988
• 2,5‐Di(tert‐butyl)‐1,4‐benzohydroquinone — a novel inhibitor of liver microsomal Ca²⁺ sequestration
  FEBS Letters, 1987
• Role of the microsomal fad-containing monooxygenase in the liver toxicity of thioacetamide S-oxide
  Toxicology, 1984
• Increase in cytosolic Ca²⁺ concentration during t‐butyl hydroperoxide metabolism by isolated hepatocytes involves NADPH oxidation and mobilization of intracellular Ca²⁺ stores
  FEBS Letters, 1984
• A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
  Analytical Biochemistry, 1976