Opening of the Mitochondrial Permeability Transition Pore Causes Depletion of Mitochondrial and Cytosolic NAD+and Is a Causative Event in the Death of Myocytes in Postischemic Reperfusion of the Heart

This publication has 40 references indexed in Scilit:

• Poly (ADP-ribose) polymerase, nitric oxide and cell death
  Trends in Pharmacological Sciences, 1999
• Fluxes of Nicotinamide Adenine Dinucleotides through Mitochondrial Membranes in Human Cultured Cells
  Published by Elsevier ,1996
• Ionic basis of ischaemic cardiac injury: insights from cellular studies
  Cardiovascular Research, 1994
• On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury
  Cardiovascular Research, 1993
• Protective Effect of FK506 on Ischemia/Reperfusion-Induced Myocardial Damage in Canine Heart
  Journal of Cardiovascular Pharmacology, 1993
• The Cell Biology of Acute Myocardial Ischemia
  Annual Review of Medicine, 1991
• Quantitative and mechanistic aspects of the hydroperoxide‐induced release of Ca²⁺ from rat liver mitochondria
  European Journal of Biochemistry, 1985
• Reperfusion of ischemic myocardium: Ultrastructural and histochemical aspects
  Journal of the American College of Cardiology, 1983
• Oxygen-induced enzyme release. Assessment of mitochondrial function in anoxic myocardial injury and effects of the mitochondrial uncoupling agent 2,4-dinitrophenol (DNP)
  Journal of Molecular and Cellular Cardiology, 1980
• Paradoxical Influence of Calcium Ions on the Permeability of the Cell Membranes of the Isolated Rat Heart
  Nature, 1966