Nuclear Localization and Dominant-negative Suppression by a Mutant SKCa3 N-terminal Channel Fragment Identified in a Patient with Schizophrenia
Open Access
- 1 July 2001
- journal article
- Published by Elsevier
- Vol. 276 (30) , 27753-27756
- https://doi.org/10.1074/jbc.c100221200
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Calcium-activated Potassium Channels Sustain Calcium Signaling in T LymphocytesJournal of Biological Chemistry, 2001
- Proceedings of the Australian Neuroscience Society Symposium: Schizophrenia A REVIEW OF THE NEUROPATHOLOGY OF SCHIZOPHRENIAClinical and Experimental Pharmacology and Physiology, 2001
- Ca2+-activated K+ Channels in Human Leukemic Jurkat T CellsJournal of Biological Chemistry, 2000
- Candidate-Gene Association Studies of SchizophreniaAmerican Journal of Human Genetics, 1999
- Length of huntingtin and its polyglutamine tract influences localization and frequency of intracellular aggregatesNature Genetics, 1998
- Isolation of a novel potassium channel gene hSKCa3 containing a polymorphic CAG repeat: a candidate for schizophrenia and bipolar disorder?Molecular Psychiatry, 1998
- Cloning of the gene for spinocerebellar ataxia 2 reveals a locus with high sensitivity to expanded CAG/glutamine repeatsNature Genetics, 1996
- Small-Conductance, Calcium-Activated Potassium Channels from Mammalian BrainScience, 1996
- Apamin-sensitive Ca2+-activated K+ channels regulate pacemaker activity in nigral dopamine neuronsNeuroReport, 1996
- Varieties of Calcium-Activated Potassium ChannelsAnnual Review of Physiology, 1989