The anti-apoptosis bcl-2 proto-oncogene is preferentially expressed in limbic structures of the primate brain

This publication has 20 references indexed in Scilit:

• GAP-43: an intrinsic determinant of neuronal development and plasticity
  Trends in Neurosciences, 1997
• Protection of Retinal Ganglion Cells from Natural and Axotomy-Induced Cell Death in Neonatal Transgenic Mice Overexpressing bcl-2
  Journal of Neuroscience, 1996
• Limbic system-associated membrane protein (LAMP) in primate amygdala and hippocampus
  Hippocampus, 1996
• Distribution of limbic system-associated membrane protein immunoreactivity in primate basal ganglia
  Neuroscience, 1995
• bcl-2 transgene expression can protect neurons against developmental and induced cell death.
  Proceedings of the National Academy of Sciences, 1995
• bcl-2 Messenger RNA is localized in neurons of the developing and adult rat brain
  Neuroscience, 1994
• Neonatal motoneurons overexpressing the bcl-2 protooncogene in transgenic mice are protected from axotomy-induced cell death.
  Proceedings of the National Academy of Sciences, 1994
• Bcl-2 Prevents Killing of Neuronal Cells by Glutamate but Not by Amyloid β Protein
  Biochemical and Biophysical Research Communications, 1993
• Calbindin D-28k and parvalbumin in the rat nervous system
  Neuroscience, 1990
• Nucleotide sequence of a t(14;18) chromosomal breakpoint in follicular lymphoma and demonstration of a breakpoint-cluster region near a transcriptionally active locus on chromosome 18.
  Proceedings of the National Academy of Sciences, 1985