JNK and c-Jun but not ERK and c-Fos are associated with sustained neointima-formation after balloon injury
- 1 January 2000
- journal article
- research article
- Published by Wiley in European Journal of Clinical Investigation
- Vol. 30 (1) , 11-17
- https://doi.org/10.1046/j.1365-2362.2000.00596.x
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Regulation and function of the JNK subgroup of MAP kinasesBiochimica et Biophysica Acta (BBA) - Reviews on Cancer, 1997
- The role of proto-oncogenes in coronary restenosis.Progress in Cardiovascular Diseases, 1997
- A Novel Mechanism of JNK1 ActivationJournal of Biological Chemistry, 1997
- Targeted disruption of the MKK4 gene causes embryonic death, inhibition of c-Jun NH 2 -terminal kinase activation, and defects in AP-1 transcriptional activityProceedings of the National Academy of Sciences, 1997
- Ultraviolet Light and Osmotic Stress: Activation of the JNK Cascade Through Multiple Growth Factor and Cytokine ReceptorsScience, 1996
- Transcription factor AP-1 regulation by mitogen-activated protein kinase signal transduction pathwaysJournal of Molecular Medicine, 1996
- Angiotensin II Stimulates Calcium-Dependent Activation of c-Jun N-Terminal KinaseMolecular and Cellular Biology, 1995
- Regulation of differentiation of vascular smooth muscle cellsPhysiological Reviews, 1995
- Smooth muscle cell immediate-early gene and growth factor activation follows vascular injury. A putative in vivo mechanism for autocrine growth.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1993
- The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformationBiochimica et Biophysica Acta (BBA) - Reviews on Cancer, 1991