Nurr1-null heterozygous mice have reduced mesolimbic and mesocortical dopamine levels and increased stress-induced locomotor activity

This publication has 35 references indexed in Scilit:

• Dopamine neurons heterozygous for the Nurr1‐null allele have reduced survival in vitro
  Neuroscience Research Communications, 2002
• Interactions Between Monoamines, Glutamate, and GABA in Schizophrenia: New Evidence
  Annual Review of Pharmacology and Toxicology, 2001
• Dysfunctional brain dopamine systems induced by psychotomimetic NMDA-receptor antagonists and the effects of antipsychotic drugs
  Brain Research Reviews, 2000
• Reduced Nurrl Expression Increases the Vulnerability of Mesencephalic Dopamine Neurons to MPTP‐Induced Injury
  Journal of Neurochemistry, 1999
• Developing a Neuronal Model for the Pathophysiology of Schizophrenia Based on the Nature of Electrophysiological Actions of Dopamine in the Prefrontal Cortex
  Neuropsychopharmacology, 1999
• Dopamine Biosynthesis Is Selectively Abolished in Substantia Nigra/Ventral Tegmental Area but Not in Hypothalamic Neurons in Mice with Targeted Disruption of the Nurr1 Gene
  Molecular and Cellular Neuroscience, 1998
• Re-evaluation of the role of the dopamine transporter in dopamine system homeostasis1Published on the World Wide Web on 27 January 1998.1
  Brain Research Reviews, 1998
• Targeted disruption of the tyrosine hydroxylase gene reveals that catecholamines are required for mouse fetal development
  Nature, 1995
• The influence of dopaminergic A10 neurons on the motor pattern evoked by substantia nigra (pars compacta) stimulation
  Behavioural Brain Research, 1989
• Dopamine receptors and the dopamine hypothesis of schizophrenia
  Synapse, 1987