Mediation of Nerve Growth Factor-driven Cell Cycle Arrest in PC12 Cells by p53
Open Access
- 1 December 2000
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 275 (48) , 37829-37837
- https://doi.org/10.1074/jbc.m003146200
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- p21WAF1 induces permanent growth arrest and enhances differentiation, but does not alter apoptosis in PC12 cellsOncogene, 1998
- Established cell lines transformed by E2F‐1 overexpression contain wild‐type p53Cell Proliferation, 1996
- The role of p53 in regulating genomic stability when DNA and RNA synthesis are inhibitedTrends in Biochemical Sciences, 1995
- Neurons from mouse embryos with a null mutation in the tumour suppressor gene p53 undergo normal cell death in the absence of neurotrophinsNeuroscience Letters, 1994
- Recovery from ultraviolet-induced growth arrest of primary rat hepatocytes byp53 antisense oligonucleotide treatmentMolecular Carcinogenesis, 1994
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Growth factor signaling: Where is the specificity?Cell, 1992
- ras mediates nerve growth factor receptor modulation of three signal-transducing protein kinases: MAP kinase, Raf-1, and RSKCell, 1992
- NGF and EGF rapidly activate p21ras in PC12 cells by distinct, convergent pathways involving tyrosine phosphorylationNeuron, 1991
- PC12 cell mutants that possess low- but not high-affinity nerve growth factor receptors neither respond to nor internalize nerve growth factor.The Journal of cell biology, 1986