Ca2+-activated but Not G Protein-mediated Inositol Phosphate Responses in Rat Neonatal Cardiomyocytes Involve Inositol 1,4,5-Trisphosphate Generation
Open Access
- 1 April 2000
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 275 (15) , 10845-10850
- https://doi.org/10.1074/jbc.275.15.10845
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Fas (CD95/Apo-1)–Mediated Damage to Ventricular Myocytes Induced by Cytotoxic T Lymphocytes From Perforin-Deficient MiceCirculation Research, 1998
- Ins(1,4,5)P3During Myocardial Ischemia and its Relationship to the Development of ArrhythmiasJournal of Molecular and Cellular Cardiology, 1996
- Arrhythmogenic Action of Thrombin During Myocardial Reperfusion via Release of Inositol 1,4,5-TriphosphateCirculation, 1996
- Suppression of Ventricular Arrhythmias During Ischemia-Reperfusion by Agents Inhibiting Ins(1,4,5)P3ReleaseCirculation, 1995
- Inositol Phosphate Release and Metabolism During Myocardial Ischemia and Reperfusion in Rat HeartCirculation Research, 1995
- Inositol trisphosphate and calcium signallingNature, 1993
- Inositol trisphosphate promotes Na-Ca exchange current by releasing calcium from sarcoplasmic reticulum in cardiac myocytes.Circulation Research, 1991
- Regulation of angiotensin II binding sites in neuronal cultures by protein kinase CAmerican Journal of Physiology-Cell Physiology, 1990
- Bovine brain cytosol contains three immunologically distinct forms of inositolphospholipid-specific phospholipase C.Proceedings of the National Academy of Sciences, 1987
- Phosphatidylinositol 4,5-bisphosphate turnover is transient while phosphatidylinositol turnover is persistent in thyrotropin-releasing hormone-stimulated rat pituitary cells.Proceedings of the National Academy of Sciences, 1986