CORTISONE AS AN ADJUNCT IN THE THERAPY OF ACUTE GOUT

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Abstract
Briefly defined, clinical gout is a metabolic disorder of urate metabolism characterized by episode attacks of joint pains, with or without redness or swelling, in hyperuricemic persons. According to Harkavy1 certain allergic factors may be involved in clinical gout. He states that there often is no relationship between the amount of uric acid in the blood and urine and the degree of arthritic involvement. Paroxysms of gout may occur with or without an elevated blood uric acid. The latter may be present between attacks when the patient is completely free of symptoms. Acutely affected joints have been found to be apparently free of urates, whereas certain symptomless joints may reveal extensive deposits of urates. There may be no constant relationship between the formation of resorption of tophi and acute attacks. Harkavy1 believes that the concept that allergy may at times be a factor in the development of gouty

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