Biphasic increase in circulating and renal TNF-α in MRL-lpr mice with differing regulatory mechanisms
- 1 January 1995
- journal article
- research article
- Published by Elsevier in Kidney International
- Vol. 47 (1) , 122-130
- https://doi.org/10.1038/ki.1995.14
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Molecular cloning and expression of the fas ligand, a novel member of the tumor necrosis factor familyCell, 1993
- Colony stimulating factor-1 in the induction of lupus nephritisKidney International, 1993
- Genetic analysis of MRL-lpr mice: relationship of the Fas apoptosis gene to disease manifestations and renal disease-modifying loci.The Journal of Experimental Medicine, 1992
- Up-regulated MHC-class II expression and γ-IFN and soluble IL-2R in lupus nephritisKidney International, 1992
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992
- The lpr and gld genes in systemic autoimmunity: life and death in the Fas laneImmunology Today, 1992
- Tumor necrosis factor Characterization at the molecular, cellular and in vivo levelFEBS Letters, 1991
- In vivo treatment of (NZB X NZW)F1 lupus-like nephritis with monoclonal antibody to gamma interferon.The Journal of Experimental Medicine, 1987
- Interaction of mutant lpr gene with background strain influences renal diseaseClinical Immunology and Immunopathology, 1985
- Spontaneous murine lupus-like syndromes. Clinical and immunopathological manifestations in several strains.The Journal of Experimental Medicine, 1978