Hemolytic Anemia in Wilson's Disease
- 23 February 1967
- journal article
- research article
- Published by Massachusetts Medical Society in New England Journal of Medicine
- Vol. 276 (8) , 439-444
- https://doi.org/10.1056/nejm196702232760804
Abstract
JAUNDICE is a common presentation of Wilson's disease and may precede the neurologic features by many years.1 , 2 Infective hepatitis is often invoked as an explanation of the initial episode, and Wilson's disease is rarely considered at that time. Subsequently, neurologic symptoms may lead to a correct diagnosis when such features as jaundice and ascites are attributed, in retrospect, to the cirrhosis of Wilson's disease. After this initial episode no further features suggesting hepatic involvement may appear despite neurologic abnormalities of many years' duration. Furthermore, ascites, of grave prognostic significance in most forms of cirrhosis, is often an early feature in . . .This publication has 14 references indexed in Scilit:
- Acute copper sulfate poisoningThe American Journal of Medicine, 1965
- Studies on Chronic Copper Poisoning:British Veterinary Journal, 1963
- Wilson's Disease: The Presenting SymptomsArchives of Disease in Childhood, 1962
- The metabolism of copper and Wilson's diseaseProceedings of the Nutrition Society, 1962
- Hämolytische Krisen als Frühmanifestation der Wilsonschen KrankheitZeitschrift für Neurologie, 1959
- Studies on Copper Metabolism. XIII. Hepatolenticular Degeneration1Journal of Clinical Investigation, 1954
- STUDIES ON COPPER METABOLISM. IX. THE TRANSPORTATION OF COPPER IN BLOOD 1Journal of Clinical Investigation, 1953
- THE COPPER AND IRON CONTENT OF BRAIN AND LIVER IN THE NORMAL AND IN HEPATO-LENTICULAR DEGENERATIONBrain, 1948
- SOME ASPECTS OF HEPATOLENTICULAR DEGENERATION AND ITS PATHOGENESISActa Psychiatrica Scandinavica, 1934
- HEPATO-LENTICULAR DEGENERATIONBrain, 1925