Lysosomal release of Cathepsin D precedes relocation of Cytochrome C and loss of mitochondrial transmembrane potential during apoptosis induced by oxidative stress
- 27 December 1999
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 27 (11-12) , 1228-1237
- https://doi.org/10.1016/s0891-5849(99)00146-x
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Oxidative stress as a mediator of apoptosisPublished by Elsevier ,2002
- Mitochondrial control of apoptosisPublished by Elsevier ,1997
- Control of apoptosis by the cellular ATP levelFEBS Letters, 1996
- Cellular injury induced by oxidative stress is mediated through lysosomal damageFree Radical Biology & Medicine, 1995
- The role of intracellular oxidants in apoptosisBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1995
- Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.The Journal of Experimental Medicine, 1995
- Mitochondrial Modifications during Rat Thymocyte Apoptosis: A Study at the Single Cell LevelExperimental Cell Research, 1994
- Accumulation of pepstatin in cultured endothelial cells and its effect on endothelin processingBiochemical and Biophysical Research Communications, 1991
- J-aggregate formation of a carbocyanine as a quantitative fluorescent indicator of membrane potentialBiochemistry, 1991
- Segregation of MHC class II molecules from MHC class I molecules in the Golgi complex for transport to lysosomal compartmentsNature, 1991