Mice lacking secretory phospholipase A2 show altered apoptosis and differentiation with Helicobacter felis infection
- 1 April 1998
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 114 (4) , 675-689
- https://doi.org/10.1016/s0016-5085(98)70581-5
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Atrophic gastric changes in both Helicobacter felis and Helicobacter pylori infected mice are host dependent and separate from antral gastritis.Gut, 1996
- Hypertrophic gastropathy in Helicobacter felis-infected wild-type C57BL/6 mice and p53 hemizygous transgenic miceGastroenterology, 1996
- Expression of Trefoil Peptides in The Gastric Mucosa of Transgenic Mice Overexpressing Transforming Growth Factor-αGrowth Factors, 1996
- The secretory phospholipase A2 gene is a candidate for the Mom1 locus, a major modifier of ApcMin-induced intestinal neoplasiaCell, 1995
- Parasitism by the "slow" bacterium Helicobacter pylori leads to altered gastric homeostasis and neoplasia.Journal of Clinical Investigation, 1994
- Genetic identification of Mom-1, a major modifier locus affecting Min-induced intestinal neoplasia in the mouseCell, 1993
- Dynamics of epithelial cells in the corpus of the mouse stomach. I. Identification of proliferative cell types and pinpointing of the stem cellThe Anatomical Record, 1993
- Identifying and counting epithelial cell types in the “corpus” of the mouse stomachThe Anatomical Record, 1992
- Use of a Mouse Model to Examine Anti-Helicobacter pylori AgentsScandinavian Journal of Gastroenterology, 1991
- A small animal model of human Helicobacter pylori active chronic gastritisGastroenterology, 1990