Cyclin kinase inhibitors are increased during experimental membranous nephropathy: Potential role in limiting glomerular epithelial cell proliferation in vivo
- 1 August 1997
- journal article
- Published by Elsevier in Kidney International
- Vol. 52 (2) , 404-413
- https://doi.org/10.1038/ki.1997.347
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Differential expression of transforming growth factor-β isoforms and receptors in experimental membranous nephropathyKidney International, 1996
- Basic fibroblast growth factor augments podocyte injury and induces glomerulosclerosis in rats with experimental membranous nephropathy.Journal of Clinical Investigation, 1995
- Long-term treatment of rats with FGF-2 results in focal segmental glomerulosclerosisKidney International, 1995
- Mitosis and the Presence of Binucleate Cells among Glomerular Podocytes in Diseased Human KidneysNephron, 1995
- Cdc2 regulatory factorsCurrent Opinion in Cell Biology, 1994
- Mesangial cell apoptosis: the major mechanism for resolution of glomerular hypercellularity in experimental mesangial proliferative nephritis.Journal of Clinical Investigation, 1994
- p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21Published by Elsevier ,1994
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Development of tubular and glomerular cells of the kidneyKidney International, 1992
- Increased urinary excretion of C5b-9 distinguishes passive Heymann nephritis in the ratKidney International, 1989