Behavior of N-acylated daunorubicins in MDR1 gene transfected and parental cells
- 7 September 1995
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 50 (6) , 889-892
- https://doi.org/10.1016/0006-2952(95)00209-i
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- BIOCHEMISTRY OF MULTIDRUG RESISTANCE MEDIATED BY THE MULTIDRUG TRANSPORTERAnnual Review of Biochemistry, 1993
- The effect of ion channel blockers, immunosuppressive agents, and other drugs on the activity of the multi‐drug transporterInternational Journal of Cancer, 1993
- Removal of the basic center from doxorubicin partially overcomes multidrug resistance and decreases cardiotoxicityAnti-Cancer Drugs, 1993
- Cyclosporins as drug resistance modifiersBiochemical Pharmacology, 1992
- Laser scanning and confocal microscopy of daunorubicin, doxorubicin, and rhodamine 123 in multidrug-resistant cellsExperimental Cell Research, 1991
- In vitro circumvention of anthracycline-resistance in ehrlich ascites tumour by anthracycline analoguesBiochemical Pharmacology, 1990
- Internal duplication and homology with bacterial transport proteins in the mdr1 (P-glycoprotein) gene from multidrug-resistant human cellsCell, 1986
- Mammalian multidrug resistance gene: Complete cDNA sequence indicates strong homology to bacterial transport proteinsCell, 1986
- Biological and physio-chemical properties of some N-acyl-daunorubicin derivativesBiochemical Pharmacology, 1979
- Mass spectrometry ofN-acylated daunorubicin derivativesJournal of Mass Spectrometry, 1976