Genetic ablation of the tumor suppressor menin causes lethality at mid-gestation with defects in multiple organs

This publication has 31 references indexed in Scilit:

• Proper coronary vascular development and heart morphogenesis depend on interaction of GATA-4 with FOG cofactors
  Genes & Development, 2001
• A mouse model of multiple endocrine neoplasia, type 1, develops multiple endocrine tumors
  Proceedings of the National Academy of Sciences, 2001
• Menin represses JunD-activated transcription by a histone deacetylase-dependent mechanism
  Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression, 1999
• Functions of c-Jun in Liver and Heart Development
  The Journal of cell biology, 1999
• Menin Interacts with the AP1 Transcription Factor JunD and Represses JunD-Activated Transcription
  Cell, 1999
• Menin, the product of the MEN1 gene, is a nuclear protein
  Proceedings of the National Academy of Sciences, 1998
• Requirement for NF-κB in osteoclast and B-cell development
  Genes & Development, 1997
• Positional Cloning of the Gene for Multiple Endocrine Neoplasia-Type 1
  Science, 1997
• Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-κB
  Nature, 1995
• Localization of the MEN1 gene to a small region within chromosome 11q13 by deletion mapping in tumors.
  Proceedings of the National Academy of Sciences, 1990