Exogenous Smad3 Accelerates Wound Healing in a Rabbit Dermal Ulcer Model
- 1 July 2004
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 123 (1) , 229-236
- https://doi.org/10.1111/j.0022-202x.2004.22730.x
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
- Ministry of Health, Labour and Welfare
This publication has 22 references indexed in Scilit:
- Cutaneous Wound HealingNew England Journal of Medicine, 1999
- Proliferation and Mitogenic Response to PDGF-BB of Fibroblasts Isolated from Chronic Venous Leg Ulcers is Ulcer-Age Dependent11Presented in part at the Gordon Research Conference on Wound Repair July 2–7 1995 and at an IBC meeting on Fibrosis in Washington, DC, April 18–19 1996.Journal of Investigative Dermatology, 1999
- TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4The EMBO Journal, 1997
- Transforming growth factor‐β: activity and efficacy in animal models of wound healingWound Repair and Regeneration, 1995
- Ex Vivo and In Vivo Gene Transfer to The Skin Using Replication-Deficient Recombinant Adenovirus VectorsJournal of Investigative Dermatology, 1994
- Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblastsThe Journal of cell biology, 1993
- Differential Modulation of bFGF Receptors by TGF-β in Adult Skin, Scleroderma Skin, and Newborn Foreskin FibroblastsJournal of Investigative Dermatology, 1992
- The Transforming Growth Factor-beta FamilyAnnual Review of Cell Biology, 1990
- Accelerated Healing of Incisional Wounds in Rats Induced by Transforming Growth Factor-βScience, 1987
- Polypeptide Transforming Growth Factors Isolated from Bovine Sources and Used for Wound Healing in VivoScience, 1983