Expression of Wild-type p53 Is Required for Efficient Global Genomic Nucleotide Excision Repair in UV-irradiated Human Fibroblasts
Open Access
- 1 October 1997
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 272 (44) , 28073-28080
- https://doi.org/10.1074/jbc.272.44.28073
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Faculty DNA polymerase δ/ɛ-mediated excision repair in response to γ radiation or ultraviolet light in p53-deficient fibroblast strains from affected members of a cancer-prone family with Li-Fraumeni syndromeCarcinogenesis: Integrative Cancer Research, 1996
- Inhibition of Nucleotide Excision Repair by the Cyclin-dependent Kinase Inhibitor p21Published by Elsevier ,1995
- The dominating effect of mutant p53Nature Genetics, 1995
- Preferential repair of ultraviolet light‐induced dna damage in the transcribed strand of the human p53 geneMolecular Carcinogenesis, 1994
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 allelesCell, 1992
- p53, guardian of the genomeNature, 1992
- (6–4)Photoproducts are removed from the DNA of UV-irradiated mammalian cells more efficiently than cyclobutane pyrimidine dimersMutation Research Letters, 1985
- Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications.Proceedings of the National Academy of Sciences, 1979
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970