Potential Role of CagA in the Inhibition of T Cell Reactivity in Helicobacter pylori Infections
- 1 June 2000
- journal article
- Published by Elsevier in Cellular Immunology
- Vol. 202 (2) , 136-139
- https://doi.org/10.1006/cimm.2000.1654
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- The m2 form of the Helicobacter pylori cytotoxin has cell type-specific vacuolating activityProceedings of the National Academy of Sciences, 1998
- Analyses of the cag pathogenicity island of Helicobacter pyloriMolecular Microbiology, 1998
- Selective Inhibition of Ii-dependent Antigen Presentation by Helicobacter pylori Toxin VacAThe Journal of Experimental Medicine, 1998
- H. pylori virulence factorsBritish Medical Bulletin, 1998
- Gastric corpus IL‐8 concentration and neutrophil infiltration in duodenal ulcer patientsAlimentary Pharmacology & Therapeutics, 1997
- Increased Cytokine Production by Gastric Mucosa in Patients with Helicobacter pylori InfectionJournal of Clinical Gastroenterology, 1997
- cag , a pathogenicity island of Helicobacter pylori, encodes type I-specific and disease-associated virulence factorsProceedings of the National Academy of Sciences, 1996
- The Proliferation of Human T Lymphocytes Stimulated by Heficobacter Pylori AntigensImmunobiology, 1996
- The vacuolating cytotoxin of Helicobacter pyloriMolecular Microbiology, 1996
- Helicobacter pylori induces lymphocyte activation in peripheral blood culturesClinical and Experimental Immunology, 1990