Elevated Levels of Plasminogen Activator Inhibitor-1 May Account for the Altered Fibrinolysis by Keloid Fibroblasts
- 1 May 1996
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 106 (5) , 1007-1011
- https://doi.org/10.1111/1523-1747.ep12338552
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- In VitroFibroplasia: Matrix Contraction, Cell Growth, and Collagen Production of Fibroblasts Cultured in Fibrin GelsExperimental Cell Research, 1996
- Elevated Levels of PDGF α Receptors in Keloid Fibroblasts Contribute to an Enhanced Response to PDGFJournal of Investigative Dermatology, 1994
- Modulation of Collagen Synthesis by Transforming Growth Factor-β in Keloid and Hypertrophic Scar FibroblastsAnnals of Plastic Surgery, 1994
- Fibronectin and fibrinolysis are not required for fibrin gel contraction by human skin fibroblastsJournal of Cellular Physiology, 1989
- Tumors: Wounds That Do Not HealNew England Journal of Medicine, 1986
- Autocrine saturation of pro-urokinase receptors on human A431 cellsCell, 1986
- Transforming growth factor β alters plasminogen activator activity in human skin fibroblastsExperimental Cell Research, 1986
- Effect of protease binding by .alpha.2-macroglobulin on intrinsic fluorescenceBiochemistry, 1982
- A study of proteases and protease-inhibitor complexes in biological fluidsThe Journal of Experimental Medicine, 1978
- THE FIBROBLAST AND WOUND REPAIRBiological Reviews, 1968