Interleukin-1β and Glutamate Activate the NF-κB/Rel Binding Site from the Regulatory Region of the Amyloid Precursor Protein Gene in Primary Neuronal Cultures
Open Access
- 1 June 1996
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 271 (25) , 15002-15007
- https://doi.org/10.1074/jbc.271.25.15002
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Opposing regulation of amyloid precursor protein by ionotropic and metabotropic glutamate receptorsNeuroReport, 1995
- Brain synapses contain inducible forms of the transcription factor NF-κBMechanisms of Development, 1993
- Regulation of the NF-ηB/rel transcription factor and IηB inhibitor systemCurrent Opinion in Cell Biology, 1993
- Degeneration in vitro of post-mitotic neurons overexpressing the Alzheimer amyloid protein precursorNature, 1992
- NF-κB Subunit Regulation in Nontransformed CD4 + T LymphocytesScience, 1992
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991
- Relation of Neuronal APP-751/APP-695 mRNA Ratio and Neuritic Plaque Density in Alzheimer's DiseaseScience, 1990
- Expression of β-amyloid precursor protein in reactive astrocytes following neuronal damageNeuron, 1989
- Characterization of the 5′-end region and the first two exons of the β-protein precursor geneBiochemical and Biophysical Research Communications, 1989
- Expression of the Alzheimer amyloid precursor gene transcripts in the human brainNeuron, 1988