Regulation of astrocyte gap junctions by hypoxia–reoxygenation
- 24 March 2000
- journal article
- Published by Elsevier in Brain Research Reviews
- Vol. 32 (1) , 250-258
- https://doi.org/10.1016/s0165-0173(99)00086-7
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- Alterations in Tau Phosphorylation in Rat and Human Neocortical Brain Slices Following Hypoxia and Glucose DeprivationExperimental Neurology, 1998
- Astrocytic Gap Junctions Remain Open during Ischemic ConditionsJournal of Neuroscience, 1998
- Astrocytic Gap Junctional Communication Decreases Neuronal Vulnerability to Oxidative Stress‐Induced Disruption of Ca2+ Homeostasis and Cell DeathJournal of Neurochemistry, 1998
- Induction of Nitric Oxide Synthase Inhibits Gap Junction Permeability in Cultured Rat AstrocytesJournal of Neurochemistry, 1996
- Reoxygenation following hypoxia stimulates lipid peroxidation and phosphatidylinositol breakdown in kidney cortical slicesBiochemical Pharmacology, 1993
- Gap junctions between cultured astrocytes: immunocytochemical, molecular, and electrophysiological analysisJournal of Neuroscience, 1991
- Glutamate Induces Calcium Waves in Cultured Astrocytes: Long-Range Glial SignalingScience, 1990
- Calmodulin Acts as an intermediary for the effects of calcium on gap junctions from crayfish lateral axonsThe Journal of Membrane Biology, 1988
- Lactic acid inhibition of gap junctional intercellular communication in in vitro astrocytes as measured by fluorescence recovery after laser photobleachingGlia, 1988
- Decoupling of heart muscle cells: Correlation with increased cytoplasmic calcium activity and with changes of nexus ultrastructureThe Journal of Membrane Biology, 1980